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This interaction is mediated by dual binding, with FHD mainly binding to the p53 TAD and FOXO4 CR3 binding to the p53 DNA-binding domain (DBD) 38 (Figure

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Second, ferroptosis prevented by GPX4 might be one of the mechanisms that explain the relative immortal nature of cancer cells that contributed to poor prognosis in patients with GPX4-positive NSCLC in this study